Cancer cells often survive treatment by fixing the DNA damage that therapy is meant to cause. Researchers found that UNI418 can disrupt this repair ability, leaving cancer cells more exposed. When combined with a PARP inhibitor, it helped resistant cancer cells respond to treatment again. The findings point to a new strategy for overcoming cancer drug resistance.
source https://www.sciencedaily.com/releases/2026/06/260610003052.htm
Extreme coastal flooding surges worldwide as rising seas rewrite 100-year
odds
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Human-caused sea-level rise has significantly increased the frequency of
extreme coastal flooding worldwide, according to a new study led by a
Tulane Unive...
16 hours ago
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