A UCLA study in mice reveals that aging muscle stem cells accumulate a protein that slows repair but boosts survival. This protein, NDRG1, acts like a brake, preventing cells from activating quickly after injury. When researchers blocked it in older mice, muscle healing sped up dramatically — but stem cells became less resilient over time. The work suggests aging may reflect a survival trade-off rather than straightforward decline.
source https://www.sciencedaily.com/releases/2026/02/260222092306.htm
Ancient tooth proteins suggest Homo erectus may have left a genetic legacy
in people today
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For most of the 20th century, the model of human origins was a tree: with
the trunk dividing into branches, and then twigs. Each species of human
relative ...
1 hour ago
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