A UCLA study in mice reveals that aging muscle stem cells accumulate a protein that slows repair but boosts survival. This protein, NDRG1, acts like a brake, preventing cells from activating quickly after injury. When researchers blocked it in older mice, muscle healing sped up dramatically — but stem cells became less resilient over time. The work suggests aging may reflect a survival trade-off rather than straightforward decline.
source https://www.sciencedaily.com/releases/2026/02/260222092306.htm
Twisting optical fiber creates a robust new pathway for light
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Light powers everything from communications to sensing, yet even tiny
imperfections can scatter it and weaken signals. To address this, a team
led by the U...
13 hours ago
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